You are on page 1of 24

reabsorption o f

[compensat mechansim,i n hypotension→


A s u a n delectrolyte
ory , ; fluido v e rloud→ A excretion-piga r i a , i f
non-enacting
kidney
→ edema

→ R i s cproduction

" '" '" "' " " " '" ' " " " '" " " '" " " " " i "
"
' '

convertinactivewit-D(cholecalciferol)t oac tive( 1 .z s ,d hydroxy.


i
Kidneyhasanyonea-9,hydroxylaseimportanti nactivationcfuitD " " " '" "
µ

↳ ✓ i t .D importantf o rA
absorptioni nc o n , s opatientw i t h re n a lfailure.→ n o secretiono fa - g draylose
h,
3
www.pti.-← fact,:-b....
m................. o fr i b - D

- o
↳i n hypotension,o d i a r r h e avomiting
r .

→ enhanceabsorptiono f watera n delectrolytes


-

"ii:÷÷:÷÷:÷÷
'" ' " " " " ' '
"
"
" ' '

atrophy(small)

°
÷÷...... sireeratiation.

↳ important becauseglomeruli i ncortexs o b yglomenalitherew i l lFilteration s on o c o r t ex n o filteration

↳ destruction>s o% calledabnormalr e n a lfunction


a n du r e aKreutinin..elevated
" " P """
""
supatiatuitucreb
" ° " " ""
e " '
" "

turn.
'


"' " "" " ' "" ""
ffiik.ru#in "£

output
becauseglamorrib e a m .
edematous f i b r o s e s - ↳filteratic,

↳ Most o fi t absorbed i n tubules, s o u r i n e outputdailyf ro m2 - 3liter e - s o o - s i t e -


i nh o tweather, w h e ntherei s destructioninglomeruli→ n i g u r i n ,destructioni ntubules→ polyurin
becausewaterfilteredb y glare-Hireabsorbed b ytubules
-

edematous,
g - clinically
→ oligurin, polyuni,i , put.-out
effusion, hypocalcaemia,a n e m i a
Leyedema,pleural
↳ investigation affectedi nmuscular panelbuilderscroutininei s
pen..,;
nine,
creutinin.i l. a w ,
highb u t t h i pnatient
i n

P Z > u re - affected
chewyprotein,
by diet
↳Normal o - t - M Y @ bleeding,
+ u r e a2.-Yong a n du p p e r↳
depend o n baseline o fpatientl i k e
croutinine steroidss ow e doped
patienti sbungcmuscular, the
2 .s o→
baselinei si .f w h e nbecome
consider o nc re a t i vei sm o r e
accurate
renalproblem
FEGER
→ to estimate 6¥12,%crFunction -Plaiting a n d f o r
cff2c.mil Failure,N o r m a l G F 12 1 2 0 £ 2 0
staying
@

because
s
females Normally have
Lower C ✓ cutininc. t h o , male,

because t h y a r t L e s , bulkyo r

Muscular
→ simple investigation, foundeverywhere
contaminated
↳ u r i n e should b e Nom o r e than 3h-u-s because. i t w i l l b e
sample

concentrated a n dsawnamount
patientfasting→ u r i n e .i s
I i rated d o i n orange

got
urine
whisti s h t oyellow

r s Cole,

↳also m yglobulinc a u s ei ti n c a s eo fmuscledestruction↳ coke-like colorl i k ei npatientw i t hpost-strop.gkmeukuephu.fi.p ro , t w i t h


c .

teau::::
= \ h kraguintor

U T I , exercise, heartfailure b u t
N o texceed Soong1dg,b u ti n destructiono fglomeruli>3-Sy
=
µ ↳slightelevationb u tn o t reaching9g-

tubulardestructioni acutetubularnecrosis,acuteinterstitial nephritis


51gb u t → i n n

n o treachingnephriticsyndrome. A albumin 2 . - 3 .m y1dg


Aalbuminei n diabetic
(micrombuminurias
nephropathy

o liguria→ 9 specificgravity, polyurin→ b specificgravity normalspecificsuit,


1-cos I - us o -

alkaline → bacterial infect.'" ↳ E c. o l i ,psi.-Sumana.


i n

acidic→ heavyproteini n t a ke , Renal tubular acidosis

(casts)

nephritis ceuaigtg.us?;rsione
casta r e m u c oprotein
incorporatedw i t hR B C
cruise,r e dglomerularcast→ glomernio nw
microscopic,mama.-www.u

nephritis,
normally From1 2 , >z infection g re-nenaturin,>
. s e a→
infection u t t e rpyelo →
f . pusc e l→l →
→ hyperKulemin i n destruction o f kidney

→ anemia, infection-Leukocytes i s

disease s oi t arrhythmia
electrolytedisturbance i n kidney
c a n cause
• Ctr , F cG →
↳f l u i doverload
underlyingc a u s e : collagendisease
fest f o r

→ Lupus Nephritis
"" " ' " " ' " '" ' "
"

a n d c o r t i c a l thickness
t.na.ms#...........
useless, i t replacedby U s , C f sc o n
→ N o wdry,

- , c y g % h e ph r o t o x i c ,s o c .I
t o d oI v p i n patient w i t h
eleyqt.eublood
→ identify stones,anyobstruction,tumor,cyst
a n d cyst a n d infarct.-c-
→ i t differentiate between tumor

# ↳ r e n a l arterystenesis u r r e n a l v e i n thrombosis
@


t.tn'" '
" '" "' ' "" ' "
' ' "
"

↳ ,
under local
. . . .". s i .

immunofluorescent
→ can't k n ow i t i s pro-renal
o r p o s t - ro n , I c a n tc

s
-

↳> 3-Sy protein June U S → Nc.-I

"I]]
"
' ""
" '" " " " "" ' '" " "

" " '" " " " '" ' "

recurrent
attack-f
u r

N e p h-et.i

↳ b-€
h o w t o know thishematuriafromkidneyo r bladder?

From kidney Fragmented,Frombladder → n o tfragmented


b ymicroscope

suif

bladder→ i fFromkidneya n disolated
n o n e e df o rbiopsy,
F rom
a n dunknown undo. big
causem e a n s u s
i snegative.E w
→ benefiti n staying u ppatient , responseo f treatment i f i tprogress t ohigha stage o f
<" "" ' " ' " ' "t" stones,t u m o r,cyst,
f .H o f c R f , a n dpatienth a scloakedcretin
- p
i n l a v a a n dr o t t e n H
, tv
a n dn o tobviousc a u s e sooner b i n y l i k ei n uncut
syndrome
" ' ""
multiple sub}?},},'"'
especiallyI , a nephropathy
a n dFabryt o diagnose
these
diseases bipy "'"
presentw i t hrecurrent

↳ t oknowc a u s e o frejectioni si t
humano rcellular?
@
'" ' " "" " " " "
→ " " ' " " " " "

biopsy

blooding r i s k

→ M u s t Lowe , B p

-
D
y

→ because therei s N o h e r oA t
b ec a s e there i s Fibrosis
→ risk f o r rupture

(relative contraindication)
-
→ r i s k F o r septico n . - u
MY

You might also like