Professional Documents
Culture Documents
Doctor, what is
definition of stroke ?
DEFINITION
The WHO defines stroke as
Types of stroke ?
CLASSIFICATION OF STROKE
Pathophysiology
Hemorrhagic strokes
are caused by a rupture of
vessels in the brain and
secondary bleeding into
brain tissue.
• Ischemic strokes can undergo hemorrhagic transformation and turn into
hemorrhagic strokes.
Ischemic Hemorrhagic
Subarachnoid
Type Thrombotic Embolic Lacunar Intracerebral (ruptured
(hypertensive) aneurysm)
Incidence 60% 20% 5% 10% 5%
Factors Occurs when
associated Occurs during Occurs while the patient is Occurs during
with onset sleep awake calm and activity
unstressed
Perfusion Small lesion
Major failure distal to seen mainly: From ruptured
causes / site of severe putamen, Hypertension aneurysms and
etiology stenosis or Due mainly to pons, vascular
occlusion of cardiac source thalamus, malformation
major vessels caudate,
internal
capsule
Gradual onset
Presentation Slowly Sudden, Abrupt or or sudden
progressive immediate gradual onset onset of local Sudden onset
deficit deficit neurologic
deficit
Another
classification ?
CLASSIFICATION OF STROKE
Time of stroke
- TIA
- RIND
- Stroke in evolution
- Complete stroke
CLASSIFICATION OF STROKE
Stroke
The Oxford Community Stroke Project
classification (OCSP, also known as the
Bamford or Oxford classification) relies
primarily on the initial symptoms
To understand bamford’s clinical
classification, you should know about
Circle of Willis
Bamford Clinical Classification
Total Anterior Circulation Syndrome (TACS)
Partial Anterior Circulation Syndrome (PACS)
Posterior Circulation Syndrome (POCS)
Lacunar Syndromes (LACS)
Neuroanatomic location of ischemic stroke
ARTERY MAIN PROBLEMS
MCA
Neuroanatomic location of ischemic
stroke
ARTERY MAIN PROBLEMS
Source : Braddom
Neuroanatomic location of ischemic
stroke
SYNDROMES LOCATION
ARTERY MAIN PROBLEMS
• Ipsilateral hemiataxia
• Ipsilateral loss of facial pain &
WALLENBERG’S Lateral medulla temperature sensation
• Contralateral loss of body pain &
temperature sensation
• Nystagmus
• Ipsilateral Horner’s syndrome
• Dysphagia & dysphonia
Source : Braddom
Lacunar Strokes
Source : Braddom
much of the benefits in mortality appear to relate to
prevention and/or earlier recognition of medical
complications of stroke and earlier mobilization
The timing and progression depend on the patient’s
condition. These activities should begin as soon as
possible (generally within 24 to 48 hours of
admission) unless the stroke survivor is unresponsive
or medically/neurologically unstable
How to manage PMR
program to a stroke
patient ?
PMR Management in a stroke patient
Acute phase
Sub acute phase
Chronic phase
Source : Module
ACUTE PHASE
Stroke
Goals :
• Prevent complications of stroke
• Prevent complications of immobilization
ACUTE PHASE
What can I do ????
Intervention PMR in acute phase ?
- Maintain skin integrity
- Prevent arise sinergic pattern and spastic ↑
- Prevent joint stiffness and shortening of the muscles
- Prevent cardiorespiratory complication
- Contend swallowing function disorder and prevent
aspiration
- Management of communication disorder
- Bowel and bladder management
- Multisensory stimulation
Source : module
Rehabilitation during the acute post stroke phase
Evaluate & manage medical problem
Monitor and adjust medication
Maintain hydration & nutrition
Facilitate rest and sleep
Venous thromboembolism prophylaxis
Proper bed and chair positioning
Frequent turns & position changes
Range of motion exercises
Deep breathing and cough exc
Frequent skin inspections
Swallowing evaluation
Safety measures
Removal of indwelling catheter, if possible, with planned, timbed
Bowel evacuation regimen
Sitting in chair
Supervised bedside exc
Self-performance of ADL
Mobilization exc
Standing & gait training as able
Educational program on stroke, recovery, and personal care
Communication evaluation and training
Psychological support to the patient
Family education and support
Source : Braddom
SUBACUTE PHASE
Goals :
- Optimally neurologic recovery and
reorganization process
- Minimize and prevent complications
Intervention PMR in subacute phase ?
- Continue the acute phase intervention
- Swallowing function therapy
- Therapeutic exercise :
1. Muscle reeducation approach
2. Neuro-facilitation approach
(Bobath, PNF, Rood, Brunnstrom)
3. Conductive educational approach
4. Motor learning approach
5. Strength training and physical conditioning
Intervention PMR in subacute phase
6. Constraint-induced movement therapy
7. Body-weight support treadmil training
8. Cognitive perceptual therapy
9. Visual imagery approach
- Electrotherapy
1. Electrical stimulation
2. Biofeedback
- Cardiorespiration fitness therapy
- Orthotic
- Assisted walking aids / ADL aids
Intervention PMR in subacute phase
- Emotion counselling
- Sexual counselling
- Educate to prevent complications
- Group therapy
CHRONIC PHASE
Goals :
- Optimally functional ability
- Maintain functional ability that had been
achieved
- Prevent complications
- Optimaly quality of life
Intervention PMR in chronic phase ?
Adaptation training
Revocational training
Counselling and education for resocialization
Home program to maintain :
- prevent joint stiffness and shortening of the muscles
- fitness training
- activity daily living according to independence level
Education and training to family/care giver to prevent
complications
Complications ?
Bronchopneumonia
Ulcer pressure
Shoulder subluxation
Shouder hand syndrome
Osteoarthrosis
Osteoporosis
Neuropatic pain
Dementia
Prognosis ?
Depend on :
Large and location of the lession
Comorbiditas
Complications
Motivation of the patient
Familly support
Facility dan professional terapist
How we can predict patient
outcome?
National Institute of Health Stroke Scale (NIHSS)
NIHSS is a systematic assessment tool that provides a
quantitative measure of stroke-related neurologic deficit.
• Other more detailed outcome measures include the Barthel Index, the
Glascow Outcome Score, etc.
Modified Rankins Scale
What is
Neuroplasticity?
Neuroplasticity refers to the ability of the central
nervous system to reorganize and remodel, articularly
after central nervous system injury.
Possible mechanisms of neural plasticity con
tributing to functional recovery might include:
dendritic sprouting over time,
new synapse formation, and
the processes of long-term potentiation and depression
Unmasking of secondary neural circuit
Source : Braddom
Clinical Application Neuroplasticity
in Rehabilitation