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  • Pathophy of Aortic Stenosis

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    Benedict James Bermas
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    A 72-year-old patient is experiencing aortic stenosis caused by the wear and tear of the aortic valve over many years resulting in fibrosis and calcification. This stenosis impedes blood flow through the aortic valve, forcing the left ventricle to contract harder to pump blood through. Over time, the increased pressure and workload on the left ventricle causes it to hypertrophy and leads to decreased cardiac output and pulmonary congestion, presenting as dyspnea, fainting, and crackles upon exertion or inspiration.

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    pathophy of aortic stenosis

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    A 72-year-old patient is experiencing aortic stenosis caused by the wear and tear of the aortic valve over many years resulting in fibrosis and calcification. This stenosis impedes blood flow through the aortic valve, forcing the left ventricle to contract harder to pump blood through. Over time, the increased pressure and workload on the left ventricle causes it to hypertrophy and leads to decreased cardiac output and pulmonary congestion, presenting as dyspnea, fainting, and crackles upon exertion or inspiration.

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    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
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    6 views1 page

    Pathophy of Aortic Stenosis

    Uploaded by

    Benedict James Bermas
    A 72-year-old patient is experiencing aortic stenosis caused by the wear and tear of the aortic valve over many years resulting in fibrosis and calcification. This stenosis impedes blood flow through the aortic valve, forcing the left ventricle to contract harder to pump blood through. Over time, the increased pressure and workload on the left ventricle causes it to hypertrophy and leads to decreased cardiac output and pulmonary congestion, presenting as dyspnea, fainting, and crackles upon exertion or inspiration.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 1

    Predisposing Factors:

    Age of 72

    Wear and tear of valve over many years


    disrupts valve and endothelium and
    underlying matrix

    Fibrosis and calcification of aortic valve


    Pathophysiology Irregular flow through valve during systolic
    contraction is audible on auscultation
    Risk Factors
    Mechanism Impedes blood flow through the aortic valve
    Heart has decreased cardiac output Decreased perfusion of
    Signs and Symptoms when doing activities brain

    LV contracts harder to pump blood across the


    stenotic valve
    Faintness on exertion
    dyspnea
    Left atrium dilates and hypertrophies form as
    a result of pressure overload Forceful contraction of LV overtime will cause
    LV myocardial hypertrophy

    Cardiac output becomes more reliant on


    atrial filling of LV Stiff and hypertrophied LV with LV pressure will
    both make the LV harder to fill (diastolic
    dysfunction)
    Bibasilar inspiratory
    Atrial fibrillation develops causing severe crackles
    decrease in cardiac output
    Pressure overload in the LV causing The passing through of fluids
    pulmonary congestion through alveolar walls into air sacs

    Dyspnea

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