Professional Documents
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I Ketut Wiargitha,SpB(K)Trauma
Divisi Bedah Trauma & Bedah Akut
LUKA : TERPUTUSNYA KONTINUITAS JARINGAN
Traumatic Wound Classification
Lacerations Contusions Abrasions Combination
Enoch, Stuart & Grey, Joseph & Harding, Keith. (2006). ABC of wound healing: Recent advances and emerging treatments. BMJ (Cl inical research ed.). 332. 962-5. 10.1136/bmj.332.7547.962.
wound healing
▪ Inflammation
▪ Fibroplasia
▪ Epithelialisation
▪ Wound contraction
Factors that affect wound healing and the
potential for infection
Patient:
- Age
- Underlying illnesses or disease: consider anemia, diabetes or immunocompromised
- Effect of the injury on healing (e.g. devascularization)
Wound:
- Organ or tissue injured
- Extent of injury
- Nature of injury (for example, a laceration will be a less complicated wound than a crush injury)
- Contamination or infection
- Time between injury and treatment (sooner is better)
Local factors:
-Haemostasis and debridement
- Timing of closure
PROBLEMS IN WOUND MANAGEMENT
The most common reasons for failure of a wound to heal are:
▪ Foreign body/material in the wound
▪ Necrotic tissue present in the wound
▪ Dead space
▪ Infection
▪ Denervation
▪ Failure to create the right local wound environment (choice of dressing
material)
▪ Host factors/disease
▪ Lack of available skin/tension on wound edges
TREATMENT PRINCIPLES
▪ Remove any barrier to healing
▪ Create an environment that supports and
encourages wound healing
▪ Repair any underlying deficiency (nutrition,
vascular or nerve supply)
STEPS IN MANAGING EMERGENCY WOUNDS--
DEBRIDEMENT, CLEANING, COVERING
▪ Thorough wound exploration--may require substantial but
gentle dissection to expose deep lacerations
▪ Remove dead, damaged and contaminated tissue
▪ Remove foreign bodies
▪ Arrest haemorrhage
▪ Restore structural normality where possible
▪ Provide drainage of dead space
Goals of Wound Care
▪ Facilitate hemostasis
▪ Decrease tissue loss
▪ Promote wound healing
▪ Minimize scar formation
Evaluation of Wounds
• Achieve hemostasis
OUTCOME:
Viable/vascularized wound bed
Decide appropriate treatment
OUTCOME:
Viable wound bed
Decide appropriate treatment
OUTCOME:
Optimal moisture balance
Decide appropriate treatment
▪ Chronic wound
▪ Prolonged inflammation
T-I-M ISSUES
WOUND CLOSURE
NPWT
OUTCOME:
Epithelialized wound
TIME Clinical Decision Support Tool
Wound Closure
• Primary closure
– Suture, staple, adhesive, or tape
– Performed on recently sustained lacerations: <12
hours generally and <24 hours on face
• Secondary closure
– Secondary intent
– Allowed to granulate
• Tertiary closure
– Delayed primary (observed for 4-5 days)
Suture Material
• Absorbable
– Chromic gut
– Vicryl
– PDS II
• Non-Absorbable
– Silk
– Prolene
– Dermalon
• Monofilament vs. braided
Staples, Adhesives & Tape
• Staples
– Quick, poor aesthetic result
• Adhesives
– Dermabond- painless, petroleum dissolves
• Tape
– Steri-strips
Pembalutan luka
Fungsinya :
• Proteksi, dari invasi bakteri (24 jam)
• Menekan, mencegah perdarahan.
• Absorbsi ( darah, transudat, exudat)
• Rasa aman , tidak takut melihat luka/ benang
jahit
• Membatasi gerak
• Memonitor perdarahan , exudasi, pus
Teknik membalut pasca bedah
Fungsinya:
▪ Drainage “Dead Space”
▪ Drainage pus,exudat infeksi/pembedahan dengan pus / infeksi
▪ Monitor adanya perdarahan yang aktif
▪ Monitor terhadap kemungkinan kebocoran anastomosis usus, dll
▪ Drainage agar terjadi “symphysis” kulit dan lapangan operasi
Asal cairan drain
X
Take-home
messages
Take-home
messages
FACTORS AFFECTING WOUND HEALING
LOCAL FACTORS SYSTEMIC FACTORS
▪ Inadequate blood supply ▪ Age
▪ Wound dehiscence ▪ Obesity
▪ Infection ▪ Malnutrition
▪ Excess local mobility joint ▪ Smoking; alcohol
▪ Poor surgical technique ▪ Diabetes mellitus
▪ Increased skin tension ▪ Malignancy; chemotherapy & radiation
▪ Foreign body; hematoma ▪ Immunosuppressant; anticoagulants
▪ Shock
DO NOT STRANGULATE
Remove foreign bodies & devitalized tissue
Achieve hemostasis
FOLLOW UP
Tulle dressing
▪ Wound contact layer paraffin
▪ Lacerations, abrations, skin loss, skin grafts, burns
▪ Chlorhexidine acetate antiseptic
▪ Framycetin sulphate antibiotic
Hydrogel
▪ Autolysis debridement & granulation
▪ Maintain a moist environment
▪ Diabetic ulcer, pressure sore; exudating or dry
necrotic wounds
CADEXOMER Iodine
▪ Necrotic tissue, slough
▪ Absorb fluids, removing exudate, pus and debris
▪ Iodine slowly released; killing micro-organisms
and forming a protective gel over the wound
surface
ANTIMICROBIAL dressing
▪ Silver dressing
▪ Broad spectrum of non-selective antibacterial action
▪ Reduce wound bacterial colonization and infection
improve healing process
Calcium alginate
▪ Effective barrier to microbial penetration for moderate to heavy LEVEL of
exudate forms a soft, gel that absorbs when it comes into contact
▪ Moist wound management
▪ Pressure sores, arterial, venous and diabetic leg ulcers, fungating lesions
▪ Hemostasis for minor bleeding
calcium ions
gel serves as a matrix for aggregation
of platelets and erythrocytes
Foam absorbent dressing
▪ Absorb exudate transpire
▪ Create a moist wound healing environment
▪ Chronic and acute exudative wounds, pressure
ulcers, diabetic foot ulcers, infected wounds,
oncological Wounds
Negative pressure wound therapy
▪ Stimulates development of granulation tissue formation and
angiogenesis
▪ Promotes moisture balance in the wound bed
▪ Removes exudate from the wound site
▪ Helps to increase vascular perfusion
▪ Edema reduction
THANK YOU