•

Heart

40*2
DERMATOLOGY PYQ’s by Dr Sara
B
? - ¥ .

,
 3
Layer
's
of the Epidermis
→ nucleus ,
flat cells

JWckest-yerz-l.IO?:: . . -inai-e
Epidermal turnover time :

Desmosomes

-22
Aug
 Cells of the Epidermis

1) Location Stratum spinosum

Longer han cells : :

BM
Origin
:

Hallmark : Beer beck

granules
→
☆ Antigen Presenting cells Pnagoyto
Markers : CD ta i LD 207

→13
langhans →_TB
98

2) Metan location Bosak

ocyte
: :

QPY
Neural crest
Origin
:

Hallmark :-) Melanin

melanin unit
Epidermal
↓
btw K.fm
association
Molas ones am ! 36k
 cell 8- Ectoderm
3) Merkel
origin
location : 8. Beas.de

slow
Type adapting
touch
-1

receptors

er

Pyd Type
-
a Rapid adapting
C) pacinian corpuscles → vibration

Auga→
Which of the following are type 1 slow adapting touch receptors?

Meissner corpuscle

→
Pacinian corpuscle
Langerhans cells
type rapid-uibr-at.org
Merkel cells

t
A 31-year-old farmer presented to the OPD with complaints of diarrhoea,
dermatitis. He further talks about having relapses in his memory. He eats

4]
maize regularly. What is the most likely diagnosis?

Beri beri → Bi
Kasal's
Wernick’s encephalopathy B.
-

% Necklace
Pellagra
Dermatitis
Keratoconjuctivitis
Diarrhea
death

Which of the following is not seen the diagnosis of the image given below:

Munro micro abscess/ Kogoj

Wick hams striae

Saw tooth acanthosis

Shiny plaques

"

Munn micro
Aʰ* rhicroabsce#

.%
abscess neutrophil
collection
of cells

lncutropnils
 Necklace
appearance
→ Niacin

Dementia
deficiency
Death
Diarrhea ,

Dermatitis ,

↓
sun exposed /Photo exposed areas

area

✓ it 133 def tryptophan deficiency

is used to form
60mg of tryptophan
Niacin
igm of
synthesis of
niacin
102 ISONIAZID inhibits

from
tryptophan aciduria
Ayano
Hartnup 's Dls → loss
of tryptophan ,

used to make
tryptophan Niacin
→ is
carcinoid tumor

Niacin
serotonin →
Melatonin Serotonin
deficiency
Tryptophan
: ,
lichen plant hnaes

"
MY Dental ¥Hac
Amatgan Patch > Patch
→
plague
< 1cm ,
¥10M 71cm

mad

Of
purple prueitic
,

/ ±

papules ,
plagues
kobner 's Pheno
/

%
p*'÷: ±¥e ✗
lacy
reticular

pattern

<
 "
Nails
"
In
Ptuyogium characteristic
is
F-

feature

"

tongue
"

_
-

Ayonycia
-

I
•

pop
tent
sign
 Sawtooth
of ridges
On Histo Rete
path

Yet another well-known histopathological finding seen in lichen

planus. Histology of lichen planus shows irregular hyperplasia of the
malpighian (spinous) layer (irregular acanthosis) in a triangular
pattern giving a saw-tooth appearance
A 27 year old female is currently on treatment with-Isotretinoin. She
is planning to get pregnant. Which of the following is a side effect of
this medication?
↓
Neural tube defect
Acne
Teratogenicity

Photo onycholysis

None of the above

' '

teratogenic
should
category ✗
drug stop
,

(thalidomide OCP etc

1 month
before getting pregnant
i

Tx
for Acne

retinoids ( Adapalene)
Grade I : Topical
A B e

O
O O

Comoclones are predominant in

grade I

-
Grade# Papulesaecpredominant
Topical antibiotics (
( Topical Retinoids +
wlinduingy

Grade 111 Pustules are predominant

antibiotics
Topical Retinoids +
Systemic
(doxycycline

Grad Iv 1 Nodule-0
Cystic
acne

oral Retinoids
Identify the plant given in the image below: me
plant
.

dermatitis
Parthenium

Ginkgo biloba

Poison Ivy

Vita trifolia

A patient presents with hypo-pigmented Iss

macula over back and upper
trunk. On investigations, spaghetti and meatball appearance was seen.
Which of the following can be the causative agent?

Malassezia Fur Fur

Pytriasis
Versicolor

o
Tinea Nigra

Trichosporon

MC M .

Globosa> m.fi
-

.
 f) Scaling
Pity riasis versicolor →
different colors
Furfur
Agent globosa↳
c. : m .
>

neetpg
SCALES :
Branny fine furfuraceous scales
i

Hypo / Hyperpigmented lesions Versicolor

-

me site :
uppertrunk
f-

10%
Investigation 8 KOH Mount

and meatball appearance

spaghetti
woodlamp's : yellow F

T /t : ① A 201 es

② selenium sulfide
 →
Wood’s lamp
dQ019_
Wavelength 360 -365
: nm

woodlamp
wavelength
-

-0
Eeythlasma
→ Coral Red Finns
•oEEda•
A 50 year old male presented with lesions given in the image below.
He later developed arthritis. Which of the following is the drug of
-

choice for this condition?

Methotre
Psoriasis Chronic Tell dls
:
,
.

inflammatory

'

Bred Red
scaly
-

&s papules & plagues

☒ white scales
way
Extensor
surfaces

Auspita Sign

pinpoint
bleeding
points

Kobner 's phenomenon 1950 morphia response

line similar lesions
A-long of
trauma

Nails pits @e change)

pit
 Subunojl hyperkeratosis

wTʰ

Oil
drop sign
CpÑgÉan§e )
0 salmon patch

Histopath

P°t9munvuomicroabsc
spongiform
F-

Neutrophil:c Collection of Celts

 PREVIOUS YEARS MCQ’S (18 JUNE 2021)
DERMATOLOGY
1. A 19 year old female student presents with urticaria following intake of sea food. She
needs a non-sedating drug to give an upcoming exam time. Which is the preferred
antihistamine for this patient?
Hi

%↳edation
A. Chlorphenitamine maleate blocker
B. Promethanine hydrochloride

-=
C. Diphenhydramine
D. Fexofenadine
Ans D
Treatment of choice for Urticaria are Antihistaminics. But antihistaminic
No BBB with no sedation as
BBB
No sedation
side effect is the second generation antihistaminic i.e fexofenadine. It Has lesser side effects
than others.
-
toe city
↳ ,

2. An image is given below. What is the most likely diagnosis?

0
p loss
0

A. Alopecia areata
B. Telogen effluvium
C. Trichotillomania
D. Anagen effluvium
Ans - A
Alopecia areata (AA) is a complex autoimmune condition that causes nonscarring hair loss. It
typically presents with sharply demarcated round patches of hair loss and may present at
Hair
any age.scarring Alopecia
Non : -

A. Areata ii.Tikomania
patchy
3. A 39 year old Tmale syphilis
farmer5.presented
Capitis to the Derma OPD with the following lesion. What
.

Diffuse (moth eaten

is theAnagen
:
effewwiam.T.ae
most likely diagnosis? - A)
patterned Aqa (Hamilton pattern
'

systemic
: SLE ,
thyroid dysfunction Anagen effwnviam
↓
chemotherapy
> 3 months ,
pregnancy
Alopecia Areata stress a
typhoid

circular patch of
hair loss
complete
lesionat
A. Mycetoma TIE :
Intra

B. chromoblastomycosis steroids

C. sporotrichosis (Triamcilone
-
D. Pheohyphomycosis.
ANS - A
1
•

^
Evnieycetonn

}→ copier
linear les
 The disease that
Copper penny
shows

seabrotic bodies
: -

bodies
/ Scloretic / Medlar
chromo bcosi }

Subcutaneousl
Mycosis y sporotvichosis
linear
follow a

:*
linear pattern
pattern

in Tom
QPYQ Sclerotic bodies are seen

↳ Chromo
On Histopath

Asteroid
bodies
-☆tffggggggAB-f-B
Mycetoma is a deep fungal infection.

Bfg☆
The classic clinical triad of mycetoma is tumor or soft-tissue swelling, sinus tracts, and
characteristic macroscopic grains. Based on the grain there is Eumycetoma and
actinomycetoma. Eumycetoma is true fungal infection and actinomycetoma is true bacterial
infection. Eumycetoma is black brown with more deformity whereas Actinomycetoma is
yellowish white with less deformity.

4. A 70 year old male presents to the OPD with the lesion given in the image below. He
recalls having the lesion since two years. What is the most likely diagnosis?

Me Skin cancer

.
A. Basal cell carcinoma
B. Squamous cell carcinoma
c. Malignant melanoma
D. Kerato acanthoma
Ans-A
Basal cell carcinoma is also known as Rodent ulcer. Nodulo-cystic variety is the commonest
variety.

5. A young lady presents with lacy lesions in oral cavity and her proximal nail fold has
extended onto the nail bed. She has a history of dental amalgam. What is the likely
diagnosis?
A - Psoriasis
B. Geographic tongue
C. Lichen planus
D. Candidiasis
ANS -C
.
Lichen planus - White lacy lesions and dental amalgam are very precipitating features of
Lichen Planus

2
 cauliflower
→
6. Identify the condition in the image?
I *•ale

%E
A. Condyloma acuminata
B. Bowen disease
C. Condyloma lata
D. Hemorrhoids
ANS-A
Condyloma acuminata - Causative agent is HPV 6, 11 and also known as genital wart.
Dont confuse between condyloma Lata and acuminata- Acuminata is warty whereas Lata is
flat topped.

7. A patient presents to the OPD with the following lesion. What is the most likely
diagnosis?

s
.
A- Hypo melanosis of Ito
B. Vitiligo (Segmental)
C. Nevus of ito
D. Becker's nevus
ANS-B
Segmental vitiligo: It follows the dermatome whereas hypomelanosis of Ito follows
blashkoid line. Blashkoid line are the migration line during embryogenesis.
Whereas dermatome is the area which has its separate nerve supply, blood supply and
lymphatics. Segmental vitiligo does not cross the midline.

3
 Ñ¥P1YMAaNTFr◦•in→①¥
melatonin
Tasty
Hypo pigmentation atom"

Albinism

defect
:
Tyrosinase
-

No
of melanocytes ⑨
↳
ueetpg

Piebabdism
crest
Defect
: N .

lesion : white forelock

Nevus
Depigmentosus
1
melanocytes
↓ melanosomes

36
keratinocytes
Defective melanosomes
 Vitiligo Acquired
-

mechanism : AI

Mc :
Vitiligo vulgaris
CIF
pg lfmge
-

☐
pigmented absence
macules

④kobnois
phenomena
of Melanocytes
kobners
phenomenon ⑤ in :
1) Psoriasis
2) L Planus
.

3) vitiligo

Bb nod

↳
Melas ma

Brown
site
symmetric macules
Malar mandible G-aced
: Nose / /

Pentz
Joyner syndrome A⑦ Pd

melanocytes
in
-
Mucosal terntiojenes →

HamartomatoOS AI
Polyps
-

to

Beckers News

Acquired condition
unilateral
Ñg mental

Hypertrichotic
L siteÉnd upper
shoulder
Dermal (color :
melanocytesis Blue to state
grey)

Mongolian spots
seen in
infants
usalllly in

ICU mbo sacral

region

nevus of Ota

unilateral ,

involves and division

Trigeminal
is &
of
Nerve
Site :
Face Serratin 213 pts)
.

News

¥
N.it#eaeiaal

PAN
fire staph ,
Streit
 -

Identify the organism causing the following lesions:

Gram positive Cocci cm → reside
Gram negative Cocci
Gram positive Bacilli
Gram negative Bacilli
!ae

Bacilli
Impeti g
Diagnosis
↓
Infection
Non-Bullous Impetigo -
Bacterial
infection of epidermis
Non bull00s : staph 1 Strep
→
GOLDEN BROWN / HONEY
COLOURED

SITE :
Face

Age group : Children

Butkus Varnish crust

Impetigo
-

aureus
staph
sub corneal
bullae

Tlt : Fosidic Acid

. or

Mupirocin
D. Cardiogenic
Ans-D Iso → Twin
Measures of central venous pressure. Central venous pressure (CVP) has a normal

All -38
range of 5–7 mm Hg in an adult spontaneously breathing patient while supine. The
me
species
CVP is elevated in obstructive or cardiogenic shock, while it is decreased in septic or
hypovolemic shock.
✗ en → Animal
33 A patient receives a kidney from his mother. What is such a graft known as?
A. Allograft
B. Isograft
C. Xenograft
Auto
D. Autograft
Ans-The transplant of an organ or tissue from one individual to another of the
same species with a different genotype. For example, a transplant from one person
to another, but not an identical twin, is an allograft.

DERMATOLOGY
1. Which of the following signs is shown in the given image below?

A. Auspitz sign
B. Demographism
C. Koebner phenomenon
D. Nikolsky sign
Ans-C

Koebner phenomenon is shown by few diseases such as lichen planus

V-vitiligo
P- Psoriasis
Koebner phenomenon is the appearance of new skin lesions on previously
unaffected skin secondary to trauma.

2. Which nail finding is the characteristic for psoriasis vulgaris as shown in image?
•

A. sublingual keratosis

36
 B. Auspitz sign
C. oil drop sign
D. On wholysis
Ans- A
Nail changes in Psoriasis
HINT-Poison
p - pitting

5-tlepifosyBTBBBLLL.BA
O -oil drop sign
B
s - sublingual hyperkeratosis
O -oncholysis
N-nail plate dystrophy

3. Identify the image.

para
.

tmitibacillary
smooth
IrE--
FIB
lifes
R
Drugs D
A. Leishmania cutanea
Rifampicin Clofamne
B. lepromatous leprosy Dapsone
c. post Kala azar dermatitis -
D. Tuberculoid leprosy.
Ans.-B
loss of eyebrows, deformity of of nose, multiple papular or nodular lesion on face.
leonine facies is characteristic of lapromatous leprosy.

%
4. Find out the best treatment for the given condition

Dager
.
✓

A. Valacyclovir
B. Imipenam
C. carbapinam
D. Penicillin
Ans-A
This is an eythematous lesion with a clear midline deposition. Herpes zoster caused
by varicella virus. It is a painful condition. Treatment given is Acyclovir or famcyclovir
or valacylovir.

5. A patient who was on anticonvulsant medication presented with rashes as

shown in the image. What could be the diagnosis?

37
 Leprosy chronic / infectious 1 Granulomatous

my
co -

leprae -

grows on
footpad of armadillo
or mice [ No culture
medium)

t⑥MgtmmityÉ No
infection
Fair
Good
1-Testable Ridley Jopling
Poor Classification
Borderline (
interchange)
1T¥ BB F- BL
tuhlstable lm-jm-F-unstab.ie
Rarest
Good
TT LL
AFB ↑ _

a-

CMI ↓
central
-
skin lesions ↑ / -1T
clearing '

#
_

Nerve involvement

Tuberculoid
leprosy (TT)
1-3-80
No
of skin lesions
-8

Annual lesion
Plague :

Central dearin§_
well
defined margins ( Nerves
]
in Anesthesia
Granulomatous disease hence
destroys nerves
resulting Alopecia
( Hair]
Boderlinetuberculoid (BT) Antrydrosis
3- 10
( skin
)
NO
of lesions :
.

special lesion : satellite lesion

periphery

gatd
seen in

> BT → satellite
unstable
 MID Borderline
leprosy CBBI
-

TT → BT -3 BB → BL-311
@
Skin lesions
Polymorphic
②
:

Geographic map like

0
Most unstable
form
0°
Borderline
leprosy
symmetrical
Almost

Skintlesions thickening
Nerve

I -¥e

lepromatous leprosy cut

symmetrical skin lesions
Nerve
thickening
Epistaxis Pedal Edema
feature
Heartiest
: .

_
•

Systemic ¢1S
 late
features inte

leonine
facies teonin facies
Entire face gets
[ I infiltrated by mieprae

Saddle nose

Dared
skin lesions I -5
bacillary Mutti
6 or
bacillary
more
-

Nerves No or IN
_
>Ñ
SSS for AFB - ⊕

montns 1
Treatment: year

}
Rifampicin 600 mg once a month reaction
lepea
Dapsone 100 milligram once daily

SEABEE
Clofazimine
+ Cloofaasauu:*
so

ngeesrpigm

Fixed eruption
Drug
intake of
drug
on re →

↓
Recurs at same site

( location is
fixed)
Hyperpigmentation
Heals E
-
-

Steven Johnson syndrome- Toxic Epidermal

Necrolysis
reaction
most severe
drug
Etiology
: >
901 .

Drug
reaction

Pathogenesis Keratinocyte : Necrosis

Body surface Area : < 101

.
CSJS
10 -
301 .
:
overlap
> 30 / TEN
'
.
.
systemic Features
CIF Atypical target lesions
: (2 zones
)
①

( proximal extremities
Features Fever , joint
systemic pain
:
,
myalgia
atrnabgia
Progress
to form Blisters →
ruptured
↓

shl tlike pidermalpe ltng-rg.TES.la or

Neetgpg →

MuC0SAliwo1mentispaominentM2g
Foul cul ITIS :
Infection of only
hair
follicle

5¥

FURUNCLE Hair
follicle surrounding area
: +

bei
follicular )

Pus point + Red tender

Timor nodule

CARBUNCLE : multiple hair

follicles
+

contiguous area

MK site :
Nape of Neck

MC association → Dm
F-

%
 A. Urticaria
B. Drug eruption
C. Angioedema
D. Cellulitis
- Ans-B
In fixed drug eruption-on drug intake there will be rash formation and it will heal
with hyperpigmentation
If drug is retake → rash will appear on same site and heal with residual
hyperpigmentation.
urticaria- edematous swelling is seen.

6.Identify the image shown below

A. Carbuncle
B .follicular abscess
C. Cellulitis
D. Erysipelas
Ans- A
caused by staph aureus
furuncle- pus forming infection of single hair follicle.
Carbuncle-pus forming infection of multiple hair follicle. Incision and drainage is the
treatment .
multiple follicle involved - yes - carbuncle if no - abscess
-

•
7. A patient presented with hypo-pigmented rash with satellite lesion as shown in
the image given below?

38
 e

A. BT

•EɧEoosso
B. BL
C. LL
D. TT

Ans-A
satellite and pseudopodia lessions are seen in BT leprosy B B-Swiss cheese or
inverted saucer appearance.

9. Identify the condition shown in the image below:

A. verrucous vulgaris
B. Elephantiasis
C. Leprosy
D. Leishmaniasis
Ans-B

FLITTERED
chronic lymphatic obstruction occurs caused by culex. in filariasis. Non pitting type of
edema usually unilateral on lower limb.

10. A truck driver presents rashes as shown in the image below. He has a history of
a lesion on his penis undergoing treatment presented with rashes. What is the
diagnosis?
STD

↳ Macedopapular
-
39
fast
A- Secondary syphilis

C. TEN →
B. Drug eruption
> 304 TBSA .

D. Steven Johnson syndrome

Ans-A

↳ COY FB ☆ A
History of lesion on penis and rashes are typically seen in secondary syphilis.
Papulosquamous rashes are seen in secondary syphilis.
.

BIOCHEMISTRY
1. Which of the following enzyme is deficient in Von Gierke’s Disease?.
A. Glucose 6 phosphate dehydrogenase
B. Glucose 6 phosphatase
C. Glycogen phosphorylase
D. Acid Maltase
Answer- B
Glycogen storage diseases - Enzyme deficient
Type 1/von-gierke's disease - glucose 6 phosphate Type 2/pompe's disease- acid
maltose
Type 3/cori's disease - Debranching enzyme
Type 4/anderson's disease- branching enzyme Type 5/ Mc arldes disease- muscle
phosphorylase. Type 6/ her's disease - hepatic glycogen

2.Complex-IV is inhibited by:

A. Rotenone
B. Malonate
C. Cyanide
D. Oligomycin
Ans- C
It is about electron transport chain
Phenobarbitone -complex 1/NADH dehydrogenase Malonate--- complex 2/succinate
dehydrogenase Antimycin --complex 3/cytochrome reductase
CO, cyanide- complex 4/cytochrome reductase Oligomycin--complex 5/F0-F1
particle.

3.Identify The Vitamin Deficiency:

40
Ans B
Aflatoxicosis occurs due to aflatoxin present in contaminated groundnuts.

29. A medical officer examines workers in mines and diagnosed them with
silicosis.What should be his comments?
A. It is reversible once we remove the cause
B. It takes 6- 10 years to develop
C. Fibrotic changes in the lungs of the patient can be removed.
D. TB screening should be done at regular intervals.

Ans. D
Silicosis, a preventable occupational lung disease, is associated with various diseases,
including tuberculosis (TB). There is an increased lifelong risk for lung tuberculosis
even if exposure to silica dust ceases

30. Long term changes/sequelae of a disease are seen in:

A. Secular trend of a disease

B. Cyclical trend
C. Disease changing its baits according to herd immunity in the population.
D. Disease changing the symptoms as per seasons

Ans - A
A disease take decades/ year to increase in a community is known as secular trend.

DERMATOLOGY

1. Auspitz's sign is seen in?

A. Pustular psoriasis.
B. Pemphigus vulgaris
C. Psoriasis vulgaris.
D. None of the above.

Ans-C.
This happens because there is thinning of the epidermal layer overlying the tips of
the dermal papillae and blood vessels within the papillae are dilated and tortuous,
which bleed readily when the scale is removed.Pustular psoriasis-no Auspitz's sign

2 First line treatment for acne comedones?

_

A.Topical steroid.
B. Topical antibiotics.
C. Benzyl peroxidase.

52
 D. Topical retinoids
Ans-D.

Grade 1- comedones-Treatment for comedones is topical retinoids ( adapalene).

isotretinoin not topical retinoids.

:¥¥÷¥÷÷÷i÷
Grade 2- Papules- treatment is topical retinoids with topical antibiotics
(clindamycin,clarthromycin). Grade 3-Pustules- treatment is topical retinoids
with benzoyl peroxide or oral antibiotics.

÷
'

*
Grade 4-nodulocystic-treatment of choice is isotretinoin (13cis- retinoic acid)
indication are severe acne,nodulocystic acne.

2. Identify the condition shown in the image?

A. Malignant melanoma.
B. Basal cell carcinoma.
C. squamous cell carcinoma.
D. nevus spilus .

Ans-A.
Basal cell carcinoma is most common showing pearly translucent nodule and
telangiectasia, rolled out edge of ulcer can also be there.
'
Evolution
Malignantmelanoma-etiology -actinic damage in genetic susceptible individual,
pathology-CDHN2A gene , clinical features are asymmetry, border,
colourchange ,diameter larger than 6mm. Best indicator of prognosis is depth.

3. Common complications associated with the following condition?

A. Post streptococcal glomerulonephritis.

B. SSSS
C. Encephilitis.
D. pneumonitis

_ET-B→T≤•EEEoᵈF7É
F-EgEÉBaÉ
Ans-A.
Non bullous impetigo - most common causative agent streptococcus than
staphylococcus, honey coloured crust seen(golden yellow).most common
complication is PSGN (post streptococcal glomerulonephritis) and that is because of
M49 strain which affects skin. Bullous impetigo-causative agent staphylococcus
aureus (ETA and ETB), varnish crust seen.

53
3. Which organism is responsible for causing erysipelas?

A. streptococcus.
B.staphylococcus.
C.Candida.
D.virus

ok
Ans-A.
Erysipelas is also called as St. Antony fire, causative agent is group ABeta
Haemolytic- STREP (only cause), upper half dermis+lymphatics involved ,Well
defined margins present (raised).

Cellulitis- both staph and strep. Are causative agent, lower half dermis and sub
.

cutis involved.

4. Cutaneous horn as shown in the image is associated with which of the

followingskin cancer?

A. Squamous cell carcinoma

B. Melanoma
C. BCC
D. Actinic keratosis

Ans-A
A cutaneous horn ( cornu cutaneum) is a hard conical projection from the skin, made
of compact keratin. They are so named as they resemble an animal's horn. They arise
from benign premalignant Or malignant skin lesions. The most common underlying
lesions are Seborrhoeic keratosis, viral warts, actinic keratosis and squamous cell
carcinoma. Horn will be painful, large size, induration at the base.

5- Identify the condition shown in the image?

A. Condyloma acuminata → HPV 6 , 11

B. Bowen disease ¥7 cervical
C. Condyloma lata

2%
D. None of the above cagers

Ans-A
g-
secondary
condyloma lata is seen in secondary syphilis and highly contagious, Broad/flat plaques
a-
seen. Human papilloma virus 6,11 is the causative agent for anogenital wartsin which
condyloma acuminata is named because condyloma acuminata is pointed growth ,
fleshy, pink and asymptomatic.
hta

54
 '

6. A female patient present with active genital warts with history of recurrence,
_ _

but her husband doesn't have it. What is the advice you will give to prevent
=
-

transmission?
A. Give continuous prophylactic antivirals to the patient.
B. Give prophylactic antivirals to the husband.
C. Give antivirals whenever active lesions are present.
D. Don't involve in sexual intercourse when a active less are present.

Ans-D
HPV that causes genital warts usually spreads through vaginal, oral, or anal sex or close
sexual contact with the genital area. Even if there are no warts, HPV might stillbe active
in the genital area and can spread to others. It is not always possible for people to
know when they got infected with HPV.

7. Patient presents with history of severe sunburn after only a few minutes in
thesun(Photosensitivity), freckling in sun exposed areas, dry skin and changes in
skinpigmentation. What is you diagnosis?
A. Xeroderma pigmentosum

umbilicated
-

=3
B. Bloom syndrome
C. Molluscum contagiosum \
D. Melanocytes nevus

Ans-A
Xeroderma pigmentosum (XP) is a very rare skin disorder where a person is
highly sensitive to sunlight, has premature skin ageing and is prone to

i-ÉBBEmgggfEsooE
developing skin cancers. Xeroderma pigmentosum is caused by cellular
hypersensitivity to ultraviolet (UV) radiation, as a result of a defect in the DNA
repair system.

Bloom syndrome is not correct because it is in herited characterised by short

stature, skin rash that develops after exposure to the sun and greatly increased
risk of cancer. Pigmentation changes seen.

8. Child presents with sores in the mouth and rash on the hands and feet as shown
inthe image below. What is the causative organism for it?

µ
A. Coxsackievirus A16
B. Pox virus
C. Herpes virus
D. Human papilloma virus

Ans. A
coxsackievirus is enteroviral infection. Clinical feature is oral exantham the andexantham in
form of papulo vesicles in palms, soles and buttocks will be seen.

55
 9. A child presents with fever and lesions which changed from one stage to
anotheras shown in the image. Diagnosis?

A. Chicken pox

B. Small pox

C. Measles

D. Rubella

Ans. A characteristic of varicella is chicken pox. 2-8 weeks incubation period. Vesicles
are red. Dew Drop on rose petal appearance seen.

ANATOMY

1. A 35 year old male presents in the ER after an RTA with fracture at the arrow
marked location. He presents with inability to extend his wrist. On examination,
there is loss of sensation dorsum of lateral aspect of hand and fingers. Which
nervewill be injured in this case?

HHV -3 ( Varicella Zoster views

A. Axillary nerve

B. Radial Nerve
chicken
C. Ulnarpox
Nerve

D. Median Nerve Inhalation of secretions

of infection
:
mode
Ans - B. and pleomorphic
Rash Centripetal
: There is wrist drop and a fracture at the shaft of the humerus . It will involve the
rose petals
nerve in the spiral groove which is the radial nerve.
Dewdrops
Wrist drop - Loss of extension of wrist.
on

In Fracture shaft of the humerus extension at elbow is intact and triceps are spared
in it

56
contact Dermatitis
irreatani (801
pts ) . non
immunological
etc
age of detergents
type I
at ˢ
Contact
Allergic
pet ×
contact

Allergic 419
dermatig _I
Atopic
Allergic Dermatitis
N
Type HS
immunologic
-

metal like nickel

seborrhoeic Dermatitis
scales
greasy yellow
Naso labial folds ⊕
C.raddle cap

w
Atopic Dermatitis

Relapsing Chronic
inflammatory
skin disease

Localised Ti Its
ATOPY :

Triad

Hallmark : intermittent
and intense
-

pruiritis

steroids
.

Ttt : Topical
skin
Emollients for dry
-