extrapyramydal system, cerebellum 3.

Muscular hypertension
*4. Hyperkinesis
Indicate structure of brain, that does not 5. Propulsion
belong to extrapyramidal system:
7. Indicate symptom, that is not
1. Caudate nucleus characteristic for lesion in pallidonigral
2. Lentil nucleus system:
*3. Nucleus of Goll and Burdach *1. Hypomimium
4. Red nucleus 2. Hypomyotonia
5. Reticular formation of brain trunk 3. Bradykinesia
4. Silent monotonous language
2. Indicate formation, that will not 5. Muscular hypertension behind
concern to paleostriatic system: plastic type
*1. Caudate nucleus
2. Globus pallidus lateral 8. Indicate symptom, that are not
3. Globus pallidus medial characteristic for parkinsonism:
4. Black substance 1. Silent monotonous language
5. Red nucleus 2. Out of flexors
*3. Hypomyotonia
3. Indicate formation, which relate to 4. Static tremor
neoeostriatic system: 5. Oligokinesia
1. Globus pallidus lateral
2. Red nucleus 9. What infringement below does not
3. Globus pallidus medial concern with hyperkinesisnes:
4. Black substance 1. Hemiballism
*5. Caudate nucleus *2. Intention tremor
3. Trochee
4. Indicate, which of the listed 4. Athetosis
formations will concern to 5. Myoclonia
extrapyramidal system of spinal cord:
1. Caudate nucleus 10. Indicate, what is characteristic of
2. Red nucleus below listed symptoms for lesion of
3. Globus pallidus caudate nucleus:
*4. Y-motoneurons 1. Muscular hypertension
5. Black substance *2. Hypomyotonia
3. Hypokinesia
5. Indicate physiological function, which 4. Bradykinesia
is not executed by extrapyramidal 5. Hypomimium
system:
1. Myostatic regulation 11. Indicate, what infringement of speech
2. Postural control arises at parkinsonism:
3. Regulation of muscular tone 1. Mutism
*4. Coordination of movement 2. Aphasia
5. Realization of automated 3. Scanned language
movements 4. Dysarthrium
*5. Silent, monotonous language
6. Indicate symptom, characteristic for
lesion in neoeostriatic system: 12. Indicate syndrome, that is
1. Hypomimium characteristic for lesion in
2. Silent monotonous language extrapyramidal system:
 1. Vestibular ataxium 2. Reticulocerebellar
2. Oligophrenia 3. Ponscerebellar
3. Dynamic ataxium 4. Vestibulocerebellar
*4. Parkinson *5. Spinocerebellar of Govers
5. Sensitive ataxium
19. Indicate symptom, not characteristic
13. Indicate basic function of cerebellum: for cerebellar disease:
1. Realization of voluntary movements 1. Intention tremor
*2. Coordination of movement *2. Muscular hypertension
3. Postural control 3. Hypomyotonia
4. Automatic movements 4. Uncertain shaky gait
5. Maintenance of expressiveness of 5. Nistagmus
mimic reactions
20. Indicate, what dissonance of
14. Indicate formation, that does not language arises at cerebellar disease:
relate with the structure of cerebellum: 1. Aphasia
1. Legs 2. Nasal language
*2. Nucleus of Goll and Burdach *3. Scanned
3. Worm 4. Dysphonia
4. Hemisphere 5. Dysarthrium
5. Dentate body
21. Which of the listed methods for
15. Indicate cerebellar nucleus: research of function of cerebellum are
1. Tail nucleus not used:
2. Red, Swalbe 1. Finger-nose test
3. Deiters, Bechterev 2. Test on diadochokinesia
*4. Fastigial nucleus, gear nucleus *3. Test of Barre
5. Nucleus of Goll and Burdach 4. Calcaneal-knee test
5. Test of Stewart-Holme
16. Indicate, which of the below-
mentioned will pass through the middle 22. Indicate, which of the below
leg of cerebellum: mentioned are afferent leading ways of
1. Dentorubralis communications of cerebellum
*2. Anterior-pons-cerebellar *1. Spinal way of Flexig
3. Rubrospinal 2. Tectospinal
4. Govers 3. Dentorubralis
5. Vestibulocerebellar 4. Vestibulospinal
5. Rubrospinal
17. Indicate, which of the below
mentioned will pass through bottom leg 23. Indicate, which of the below
of cerebellum: mentioned are efferent leading ways of
1. Spinocerebellar of Govers communications of cerebellum:
*2. Spinocerebellar of Flexig 1. Spine-cerebellar Flexig
3. Frontal bridge 2. Spinocerebellar of Govers
4. Rubrospinal 3. Vestibulocerebellar
5. Occipital bridge *4. Dentorubralis
5. Front-pons-cerebellar
18. Indicate ways, that will pass through
top leg of cerebellum: 24. Name method of revealing dynamic
1. Olivocerebellar cerebellar ataxium:
 *1. Finger-nose test 5. Peripheral tetraparesis
2. Station test
3. Test of Barre 30. On what side of cerebellum
4. Test of Budda infringements at hemilesion and cortex of
5. Liquorodynamic tests cerebellar hemispheres are developed:
1. Ataxium of extremities of opposite
25. Indicate infringement, characterized focus
by lesion of vermis: *2. Ataxium of extremities on side of
*1. Static ataxium focus
2. Sensitive ataxium 3. Ataxium of trunk from opposite side
3. Muscular hypertension 4. Ataxium of trunk on same side of
4. Bradykinesia foucs
5. Vestibular ataxium 5. Bilateral ataxium of extremities

26. Indicate detection method of 31. Impossibile to walk a straight line,

infringement of statics: alternately putting right heel of one leg to
1. Test on diadochokinesia toes of other, is connected to:
2. Test on hypermetria *1. Cerebellar dysfunction
3. Finger-nose test 2. Lesion of parietal share of brain
*4. Stability in pose Romberg 3. Lesion of temporal share of brain
5. Test of Babiski 4. Oculomotor dysfunction
5. Loss of sensitivity in cadences
27.
Name clinical attribute of cortical-frontal 32. Tremor of hands in calmness,
ataxium: especially when awakening the patient,
1. Deviation at movements aside lesion is usually developed where:
center 1. Visual hump
2. Nausea, vomitting *2. Black substance
3. Nistagmus 3. Caudate nucleus
*4. Astasium-abasia 4. Spinal cord
5. Scanned language 5. Inside capsule of brain

28. Indicate biochemical mechanism of 33. Adiadochokinesia testifies about

development of Parkinson syndrome:
1. Infringement of copper metabolism
2. Infringement of exchange of
phenylalanine
*3. Infringement of exchange of
dofaminum
4. Infringement of carbohydrate
metabolism
5. Infringement of exchange of lipids
29. Indicate syndromes, that are
characteristic for lesion of tail and red
nuclei, «оЁб®perл© body:
1. Parkinson
*2. Hyperkinetic
3. Non-tactal
4. Central tetraparesis
infringement of:
1. Consecutive movement of fingers
2. Walking from heel to sock
3. Hold back of tremor
*4. Synchronisation of movementsa of
arms
5. Coordination of colloquial muscles
34. Indicate medication, that is used for
treatment of Parkinson syndrome:
1. Nootropic preparations
2. Sedative mean
*3. Cholinoblockers (anticholinergic)
4. Anticholine esterase preparations
5. Corticosteroids
35. Sharp static ataxium is characteristic
with muscular atony and asynergia for
lesion of :
1. Bottom legs of cerebellum
2. Middle legs of cerebellum
3. Top legs of cerebellum
*4. Vermis
5. Cerebellar hemispheres
36.
Uncertainty and unsteadiness while
walking in darkness and good
illumination are characteristic for
ataxium:
*1. Sensitive
2. Vestibular
3. Dynamical cerebellar
4. Static-locomotor cerebellar
5. Cortical (frontal)
37. Functions of cerebellum are all of the
below listed, except:
1. Coordination of movement
2. Regulation of muscular tone
3. Synergy of movements
4. Equilibrium of body
*5. Postural tone
38. Indicate, with what choreic
hyperkinesis is clinically characterized:
1. Slowness
*2. Speed, unevenness, precipitancy,
absence of stereotype
3. Speed, unevenness, precipitancy,
stereotype
4. Twitching in muscular groups and
in lonely muscles
5. Involuntary tonic reduction of
mimic muscles of person
39. Patient has hypomimicry, flexor,
shuffling gait, muscular hypertension
behind plastic type. Indicate localization
of pathological center:
1. Neoeostriatic system
*2. Pallidonigral system
3. Cerebellum
4. Internal capsule
5. Brain trunk
40. Ten year old child has violent
movements of extremities face, trunk.
Indicate localization of pathological
center:
1. Pallidonigral system
*2. Neoeostriatic system
3. Cerebellum
4. Fronto-prefrontal site of cerebrum
5. Anterior central convolution
41. Patient with parkinson's disease
syndrome. Indicate, how gait is affected:
1. Spastic
2. Spastico-atactic
3. Hemiparesthetical
4. Steppage
* 5. Shuffling, fine steps
42. Due to disease unsteadiness at
circulation appeared, intensive tremor at
finger-nose and calcaneal-knee tests,
More expressed on the right side,
Nistagmus at sight to the right. In pose
Romberg patient falls to the right.
Determine localization of lesion:
1. Vermis
2. Lefthand cerebellar hemisphere
*3. Right cerebellar hemisphere
4. Top legs of cerebellum
5. Bottom legs of cerebellum
43. Fast arrhythmic involuntary
movements of extremities and trunk are
observed in patient.
He grimaces, frequently sticks out
tongue. Tone of muscles of extremities is
lowered.
Described syndrome is named:
1. Athetosis
2. Myoclonia
*3. Trochee
4. Hemiballism
5. Tic
44. Patient is worried by difficulty while
walking. Objectively -- Hypomimium,
delay of voluntary movements, increase
of muscular tone of extremities behind
plastic type, walks in small steps,
shuffling. Described syndrome is named:
 1. Hyperkinetic
*2. Parkinson
3. Non-tactal
4. Alternating
5. Brown-Sechar
45. Patient walks with short steps, body
is inclined forward, hands half-bent, face
is masklike.
Speech monotonous, silent, calmed
down. Stereotyped tremor of fingers of
hands of "account of coins "type is
observed. Syndrome is named:
1. Alternating
2. Non-tactal
3. Hyperkinetic
*4. Parkinson
5. Brown-Sechar
46. Patient after having influenza
developed violent vermiculations in
fingers of bones.
These movements are named:
*1. Athetosis
2. Hemiballism
3. Myoclonia
4. Tic
5. Trochee
47. Patient has sharp headaches, nausea,
vomitting. At objective research
horizontal nystagmus is revealed, it is
primary at sight to the left,
adiadochokinesia at the left, scanned
language, unsteadiness of gait, in pose
Romberg falls to the left.
Determine localization of lesion:
1. Top legs of cerebellum
2. Bottom legs of cerebellum
*3. Cerebellar hemisphere at the left
4. Cerebellar hemisphere on the right
5. Vermis
48. Schoolboy began to slovenly, turns
head in different directions during
lessons, grimace, write letters of different
size.
Objectively --
Lowered tone of muscles in extremities,
fast arrhythmic involuntary movements
of extremities and trunk are observed.
Described syndrome is named:
1. Athetosis
2. Hemiballism
3. Myoclonia
4. Tic
*5. Trochee
49. Man-40 years old choreolike
hyperkinesis, dementia.
Indicated symptoms gradually progress.
Name disease:
1. Sharp cerebral circulatory
disturbance
2. Paralysis agitan
3. Paraplegia of Strumpell
*4. Huntington disease
5. Meningoencephalitis
50. 36 yr old man in residual period of
encephalitis, arose attacks of myoclonia
of muscles of face, and left hand.
At review in interattack period -
somewhat lowered muscular tone in left
sided extremities, intensive tremor,
hypermetria of left sided extremities.
Indicate localization of lesion:
*1. Red nucleus on the right
2. Black substance at the left
3. Vermis
4. Lefthand cerebellar hemisphere
5. Dentate body on the right
51. Man 42 years, 3 year back survived
head trauma.
Complains on unsteadiness of gait,
infringement of movements of right
hand and leg.
At review - right-hand pyramidal
deficiency behind central type,
coordination tests at the left execute with
intention, adiadochokinesia through
negative chronotropism at the left,
hypomyotonia at the left.
Indicate striked structures:
*1. Anterior central convolution at the
left, left side of hemisphere of
cerebellum.
 2. Anterior central convolution at the
left, lefthand frontal -cerebellar way
3. Anterior central convolution on the
right, right frontal-cerebellar way
4. Spinocerebellar tracts on the right,
rubrospinal at the left
5. Spinocerebellar tracts at the left,
rubrospinal on the right
52. Woman 42 years, complains of
residual phenomena of trunk encephalitis
on complication of gait, unsteadiness,
impossible to stand in vertical position
and balance.
At review - light central tetraparesis,
bilateral expressed static-locomotor
ataxium.
Indicate localization of lesion:
*1. Vermis, overlap of pyramidal route
2. Vermis, cerebellar hemisphere
3. Cerebellar hemisphere, red nuclei
4. Internal capsule on the right, vermis
5. Internal capsule at the left, vermis
53. Patient has shaky gait, on the right
missdirect, intensive tremor,
adiadochokinesia, horizontal nystagmus.
Where can lesion be located:
1. Vermis
2. Right cerebral peduncle
3. Lefthand cerebellar hemisphere
*4. Right cerebellar hemisphere
5. Lefthand anterior central
convolution
55. Patient is worried by shaky gait,
rotatory vertigo, nausea, cannot stand in
Horizontal position.Nystagmus checks
out. Falls In Romberg pose
What is damaged?
1. Vermis
2. All parts of cerebellum
3. Back cords of spinal cord
*4. Vestibular apparatus
5. Cerebellar hemisphere
56. In woman, 42 years, appeared
infringement of movements in left hand,
general constraint.
Therapist, at which woman was treated
from psoriatic polyarthritis, suspected
neurological disorder.
Neurologic inspection found:
Hypomimicry, bradykinesia, flexor pose,
extrapyramidal hypertonus.
Pathogenetic therapy was ordered
Which medication is most used in this
instance:
1. Selegelin
*2Cyclodol
3. Bromocryptine
4.Haloperidol
5.Amantadin

54. Patient is moved with effort, cannot

stand because of unsteadiness, falls in
pose of Romberg, also cannot sit.
Volume of active movements in
extremities is full, force of muscles is 5
points.
Sensitivity (superficial and deep) is kept.
What is damaged?:
1. Weigh cerebellum
2. Back cords of spinal cord
3. Cerebellar hemisphere
*4. Vermis
5. Precentral convolution of the
brainof cortex