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Infectious Diseases of the Circulatory ● Stage 4 – AIDS

System ▪ The immune


system weakens
too much as
DISEASE ETIOLOGIC AGENT
 HIV/AIDS  Human CD4 cells
 Lymphadenopat Immunodeficiency decrease in
hy associated Virus, a unique number
virus (LAV), type of virus
 AIDS associated (retrovirus) that PREVENTION MANAGEMENT
with retrovirus invades helper T ● Abstinence ● antiretroviral ther
(ARV) cells (CD4 cells ● Monogamous apy (ART)
▪ It is  Also called Human relationship Nucleoside Reverse
preventable, T cell lymphotropic ● Protected sex Transcriptase
manageable but virus (HTLV III) ● Sterile needles inhibitors
not curable ● New ● Non-nucleoside
▪ AIDS limits the shaving/cutting transcriptase
body’s ability to blades inhibitors
fight infection ● Protease inhibitors
due to markedly
reduced helper DISEASE ETIOLOGIC AGENT
T-cell  MUMPS  Caused by
 Also called Epstein-Barr virus
NATURAL COURSE RESERVOIR AND “mono” or (EBV), which is
● Stage I – MODE OF “kissing disease” known as human
Primary TRANSMISSION  Usually self herpesvirus 4; EBV
▪ short, flu like ● Blood products limiting (4-6 wks) is known to be
illness (1-6 wks ● Semen / vaginal rarely fatal oncogenic
after infection) fluids  Complications:
▪ Mild symptoms ● Intravenous drug enlarged spleen
▪ Infected person abuse (sharing of SIGNS AND MODE OF
can infect other needle) SYMPTOMS TRANSMISSION
people ● Unsterilized blades ● Fatigue ● Through kissing
● Stage 2 – ● Unprotected sexual ● Sore throat ● Sharing of glass or
Asymptomatic intercourse (oral, ● Fever food utensils with
▪ Lasts an anal) ● Swollen lymph someone who is
average of 10 ● Mother to baby nodes in the infected
years (before and during neck and armpits
▪ Swollen glands, birth) ● Swollen tonsils
level of HIV in ● Headache
the blood drops ● Skin rash
▪ HIV antibodies ● Soft, swollen
are detectable spleen
in the blood PREVENTION
● Stage 3 – ● Hand hygiene
Symptomatic regularly
▪ The immune ● Not sharing
system food, dishes,
deteriorates glasses and
▪ Opportunistic utensils
infections and
cancers start to
appear

PILONES,RISHELLE MAE M.
DISEASE ETIOLOGIC AGENT DISEASE ETIOLOGIC AGENT
 Mumps  Dengue ● Vector: Aedes
 Known as Hemorrhagic aegypti
infectious Fever
parotitis  Known as the
 Complications: “breaking-bone
Orchitis, fever”
Oophoritis  Self limiting 6-7
MODE OF SIGNS AND days
TRANSMISSION SYMPTOMS
● via droplet ● Signs and COMPLICATIONS SIGNS AND
spread symptoms ● Intense SYMPTOMS
● direct contract develop, usually abdominal pain ● Phase 1
with the saliva appear 2-3 wks ● severe internal ▪ fever
after exposure bleeding ▪ weakness
● Primary signs are ● Circulatory ▪ edema of the
swelling and collapse extremities
tenderness of the ● death ▪ severe pain in the
salivary glands head and muscles
cause by ARV ● Phase 2
● Pain in the ▪ return of fever
swollen salivary ▪ bright red rash
glands on one or
both sides TREATMENT PREVENTION
● Pain while ● no specific ● control of
chewing or treatment mosquitoes
swallowing ● proper hydration
● Fever, headache,
muscle ache
● Weakness and
fatigue
● Loss of appetite

PREVENTION MANAGEMENT
● best way is thru ● rest as much as
vaccination possible
● try to ease
symptoms with
cold compress
● OTC pain reliever

PILONES,RISHELLE MAE M.

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