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stimulate

prostaglandin
decrease prostaglandin haemorrhage se tfiiocnacid proteolysis T hydroagihdor icy f
prostaglandins
lipolysis y
# Jalcohol (
r lesion n
inhibit activate

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aciindcrfraoseduction free radical ^
f
Helicobacter NSAID
T
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acid
pepsin
Pepsinogen
fmbphospholipid
arbonate buffer
barrier

smoking ( Pyu
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p f / y mucosal layer →

ogenloug
I increase

Agftisfnadmine /
'
endogenous hydrophobicity
stress
mucosal surface →

standard triple p f f epithelium


therapy \ Factor Defensive endothelial barrier
Aggressive →
mucosa , →

endothelial mediators
alternative bismuth based - ←
Management T factor
vasculature →

↳ mucosal blood flow


quadrille therapy L \ )
helicobacter pylori infection
Hyper plastic polyp ←
Differential Gastric → Pathophysiology

↳ arterial wall necrosis z

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Pseudoayeurysm

/ Diagnosis ↳ of

Polyp
multiplication
✓ foveola cells -

Juvenile weakened
↳ Classification
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/
L
polyp wall ruptures
Unica '
emdgmwgto.us and inflamed endoscopy
Evaluation
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Pathogenesis hey!!gg¥ss;f;÷i"
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syndrome
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→ ,
adenoma
& & Gastric carcinoid →
neoplasms of endocrine
Inflammatory Fibroid
hyper regeneration
-
polypoid and

Polyp circumscribed →
epithelium of
dysplastic
, f granuloma

ymindmap-n.gge.TL

gastric submucosa layer Azzahra D. Sophia
eosinophilic infiltration damaged mucosal lining
1806226935 -
Group F

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