2.

9 WOC

Idiopatic Urinary tract damage of immobilization dehydration or fluid Food contains high calcium,
infection tubule in nefron intake decrease oxalate and purine

bone
bacteria urea urine static movement increasing excess of calcium,
breaker less concentration in oxalate and purine
urine and organic
materials solution
Sedimentation and accumulation
cristalisation calcium increasing
proliferation
calcium in plasma of urine
increase
urine pH is acid

Cristalisation Process

Calculi Formation

NP : DEFICIT OF
UROLITHIASIS Operation/Surgery
KNOWLEDGE

Obstruction of urinary tract NP : ANXIETY

Stones at Kidney Stones at Ureter Stones at Vesica Urinaria

obstruction of irritation endotel and

blood flow blood vessel at ureter bladder bladder neck

Hematuria reflux urine urine retention

destruction of Release
inflammation to kidney
kidney tissus
NP : RISK OF mediator urine retention
INFECTION (pirogen) Hydroureter Hydronephrosis
kidney function
decreased NP : DISTURBANCE
activate
OF URINE
arachidonat kidney dilatation
kidney colic NP : ACUTE ELIMINATION
acid
PAIN PATTERN

reflex stimulate Urine Statis pressure at NP : ACUTE

reteointestinal thermostat in kidney structure PAIN
hipothalamus

nausea and kidney tubule

vomiting body dilatation
temperature
increased
NP : NUTRITION damage of
LESS THAN BODY kidney function
REQUIREMENTS NP : HIPERTERMI
CRF