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-
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.

in
secretes Melatonin mainly -

regulation
EN
Sleep wake
cycle

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-

V
o•D Body temperature
Influences metabolism

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woman .ae TiEsa÷mgiie
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eight fansite delays puberty defense capability
Third
fo
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during dark
vertebrates d
day
.

n - n
 Melatonin
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precursor
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tryptophan
Melanin

figurehead 'T
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-

avg.nu .

phenylalanine
Ab
melanin
is
Albini
-hysoidGlandJ Located both sides trachea
-
on
of
Both lobes connected
by
-
are
Thyroid
hormones Isthmus .

detentes
BI Tz Triiodolhysonine
→
→

TBD > Ty 737

tetraoriofsthomal
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-

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potent Forren Thyroxine
tissue
-
=

secrete calcitonin
T
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>
o F-

Thyrogtobulin
Hai
MY
sink
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EI
Isthmus
( connective
tissue )
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(
-

→ ÷
-
.
Para -

follicular cells
/ C -

cells 7

÷ .
÷§§§- Thyrogeoesuein
→ ( ÷
( made
of Tyrosine
%YYThyw calcitonin)
AAS)
.

Tottopirdalm J
'd H,
÷ 883¥ augur
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hormone

converted
taken
follicular cells 73 sty
I
by
TG to
up
T
I secreted
z
in blood
\ I
C H MHz
HO O Choy
-
-
-
- -
-

,
/
/ C OOH
3
I

Thyroxine .
or
⑤ Tyr + I →

Monoiodothynosnnnine
( ) MIT
+21 →
Diiodothyronine
( DIT)
Note : -

Ty secreted reaches
target cells
-

have
target cells

Iodine
I
73
Ty -
 Functions catabolic
of Thyroid
hormones o

activity
-

→
of ca#rates # proteins
d
→ Metabolism ,

Glywgenolysl

Li p ol y si s
Anabolism
's

Regulates Basalmetatsoht.RU#
→

( protein synthesis)
-

Development of mentalities
→
Metamorphosis in
frog 2

EEE
↳
→ Influences

mil ar in
-Body temp regulation

function
Toi t
→
MT 'd * blood

used
L
for
myelination
-
. .

#gon consumption T
%#
RB{ ( Erythropoiesis )
-
 thymol disorders
HYPOTHYROIDISM
HYPERTHYROIDISM
Grave 's
t
Thyroxine disease
Thyroxine T
,

q
Simple Goitre Goitre
Exopthalmic
*
*
t

m-7hyatg÷Y÷mmdpeui
Ia deficiency → Auto immune disorder
-

L autoantibodies
Thyroxine deficiency
-

I
'
TSH - are
formed
Cretinism action
) Thyroid dwarfism the
mimic TSH
*
of

heightening.in?a7aggatItyardawitn.nng
-

hypertension
*
Myx oedema / Gull 's disease Edema behind the
eyesprotrusion
→
T
-
Accumulation
" of fluid
in
facial tissues of Eyes
Face
"

puffy oEp@nos
-

Autoimmune
Hashimoto 's eye
→

* disease disorder

Thyroid Hyper
cancer
-

Antibodies
thyroid follicles
t
against
→

- Antibodies
destroy follicular cells secretion
of Thyroxine
-

Thyroxine dtd secretion

,
Hypo
 Parathyroids 4-
pea sized glands dorsal side
-

of thyroid lobes
y Tce&
Chief cells
Antagonistic pair
tcrete formed inundations
blood
parathion more
under
.

p% cakitm*yiftfEe
✓Hp hormone
's hormone
£

Hypercalcemia
g- Finished
Cat Zabsofotn
Tse
dietary
Ise Cat -
excretion
The Cath reabsorption from
tubules
Tses bone demineradisateonl resorption ( osteoclast t
activity )
 Skin ¥s mobilise

( Precursor t
Lives
Vit D )
-

of d
wit Da

ltidgeyaus
v£D3
PTH

Haloed → Intestine
L
Tse Cat 2
absorption
diet
from
 -

←jttEEE
-

a .

←
-

caiuihes
inflammation
-

J-£ seizures/fE bone

ej Cath controls the

=
Nat influx in
in bone

neurons

to
Nat influence

Ryti 'd action

potentials
wid contraction ( spasms ) .

fltypocalcaemic tetany)
 Thymuscilandf -

Primary lymphoid organ

→

ThroneofTmmumI#
-
Located behi¥n outline

←
ventral .

→
Secretes a
peptide hormone

spake b/w
Thy tosin
two
lungs
•
°

.
Training school of ⇐ responsible for differentiation
of T
lymphocytes
-

Lymphocytes
.

( Cms) L

phqgfym-isu.eu
Hassel's
corpuscles are L attain

meewatedthef.cigenic
Immunity receptors .

age ( starting from puberty )

Atrophies with
I
reduced (degenerated .
 Lymphoid
^ organs
Primary to
Secondary L o .
. -

I t
&
casein )proliferation Lymphocytes interact
w
differentiation
'

with
antigens )
of Lymphocytes pathogens .

spleen
( lymphocytes ) eg
-

T
G Thymus
-

-
Tonsils
nodes
Bone Lymph
-

marrow

↳ B
( -

lymphocytes ) -

Peyer 's patch

-

MALT

( Mucosal
Associated
50yd Lymphoid
final
Tissue )
lymphoid
tissue
 I.
Immunity
Innate
Acquired
(
By Birth ) ( lifetime )
eg skin
Mucosa
#
T
lymphocytes
B-
lymphocytes

Cgslthtufsodii es
-

Ieee
L t

lysozyme cell Mediated

Immunity
Antibody
mediated
immunity