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    Non-cirrhotic ascites is generally caused by peritoneal carcinomatosis from primary or metastatic cancers, peritoneal infections, or pancreatic disease. Treatment focuses on managing the underlying condition causing the ascites. When initiating diuretic therapy and sodium restriction for ascites, weight loss should be monitored and not exceed 0.5 kg per day for patients without edema or 1 kg per day for those with edema. If inadequate response occurs, diuretic doses should be increased and urine sodium checked to assess diet compliance. Electrolytes and kidney function should also be regularly monitored during diuretic treatment.

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    Non-cirrhotic ascites is generally caused by peritoneal carcinomatosis from primary or metastatic cancers, peritoneal infections, or pancreatic disease. Treatment focuses on managing the underlying condition causing the ascites. When initiating diuretic therapy and sodium restriction for ascites, weight loss should be monitored and not exceed 0.5 kg per day for patients without edema or 1 kg per day for those with edema. If inadequate response occurs, diuretic doses should be increased and urine sodium checked to assess diet compliance. Electrolytes and kidney function should also be regularly monitored during diuretic treatment.

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    6 views1 page

    DD 38

    Uploaded by

    GOT7 for 7
    Non-cirrhotic ascites is generally caused by peritoneal carcinomatosis from primary or metastatic cancers, peritoneal infections, or pancreatic disease. Treatment focuses on managing the underlying condition causing the ascites. When initiating diuretic therapy and sodium restriction for ascites, weight loss should be monitored and not exceed 0.5 kg per day for patients without edema or 1 kg per day for those with edema. If inadequate response occurs, diuretic doses should be increased and urine sodium checked to assess diet compliance. Electrolytes and kidney function should also be regularly monitored during diuretic treatment.

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    NON- CIRRHOTIC ASCITES

    Ascites in the absence of cirrhosis generally results from peritoneal carcinomatosis,


    peritoneal infection, or pancreatic disease. Peritoneal carcinomatosis can result from
    primary peritoneal malignancies such as mesothelioma or sarcoma, abdominal
    malignancies such as gastric or colonic adenocarcinoma, or metastatic disease from
    breast or lung carcinoma or melanoma.

    Treatment is based on management of the underlying cause.

    MONITORING

    With initiation of dietary sodium restriction and diuretic therapy

    • Aim for weight loss of no more than 0.5 kg/d in patients without edema

    • Aim for weight loss of no more than 1.0 kg/d in patients with edema

    • Inadequate response is defined as < 1 kg loss in the first 7 days and 2 kg loss
    every 7 days thereafter

    • If response is inadequate, increase the dose of diuretics

    • Urine sodium can be checked in nonresponders to see if they are adherent with a
    low-sodium diet.

    Serum electrolytes, BUN and creatinine should be

    assessed regularly to monitor for complications of

    diuretic therapy

    • Regular clinical assessment for signs and symptoms of hepatic encephalopathy


    and spontaneous bacterial peritonitis.

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